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[12] [22] The once-weekly administration and gradual pharmacokinetic profile may contribute to improved gastrointestinal tolerability

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By contrast, other studies have shown that global GIPR-KO mice are protected from both obesity and insulin resistance when fed a high-fat diet (3135), and GIPR antagonism coupled with GLP-1R agonism leads to weight loss in early clinical trials and mouse models (36, 37), possibly by preventing leptin resistance (16, 38, 39)

By regulating PEG length, non-specific tissue distribution of ADC in the organism can be ameliorated accordingly, thus effectively reducing off-target toxicity

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